Discover and read the best of Twitter Threads about #Takotsubo
Most recents (8)
It is fitting that this issues is being published soon after the passing of Dr. Marty Samuels. Among his many gifts, he was a clinical virtuoso at the intersection of medical and neurological illness. Neurocardioloy, neurohematology, neuronephrology, neurorheumatology...
In defining these fields, he brought the best out of all of us... stressing holistic patient care, collaboration, and curiosity. He imbued us with a passion for caring for conditions that fell between subspecialty cracks. I am so grateful to have learned from him...
He would have loved this issue of Continuum and no doubt would have referenced (or even argued) a nuanced point at his next Neurology Report with @Tracey1milligan . I will always treasure the complimentary email he sent me re: my first paper on his favorite topic: #Takotsubo
Otro casito bueno pa la colección, abro 🧵 Mujer 37 años PO miomectomía laparoscópica, sin APP. Refiere anestesio que se le dió AGB, se resecaron los miomas pero aparentemente quedaban restos, se volvió a irrigar con glicina en cavidad uterina para distenderla y posterior a esto
…inicia con bradicardia de 30 lpm, SAT 60% y poliuria, sangró 700, se le transfunde 1 PG, 1 L Hartmann e inicia vasopresor, la hiperventila tratando de mejorar SAT y posterior pasa a UCI. Donde la recibimos con efecto residual de sedación, RASS -4 pupilas isocóricas de 6 mm…
… con PAM 69, FC 40, FR 16 SAT 60%, con nore a 0.2 mcg/k/min, ritmo sinusal por telemetría pero con extrasístoles ventriculares aisladas, llc de 5 segs, VMI, Fio2 al 60%, csps crepitantes, abdomen con hx qx sin datos de dehiscencia o sangrado. Solo con GV en ese momento…
Glad to share our latest publication on #WhyCMR with T1 and T2 mapping in #takotsubo syndrome 😊with @lucarlimite @beamusumeci1 @c_eike @v_puntmann academic.oup.com/ehjcimaging/ad…
Myocardial edema largely represented, correlations with interstitial expansion, ECG changes and systolic function. Some considerations below 🧵
1/ In #takotsubo syndrome myocardial edema features the acute phase: #WhyCMR T1 and T2 mapping have the advantage of a parametric quantification of edema. T2 specific for water, but native T1 can read it too (ahajournals.org/doi/full/10.11…) and ECV not water-independent in this setting
😊 first-authorship on @JACCjournals❗️ @beamusumeci1 jacc.org/doi/abs/10.101… - Thankful to @FraSantoroMD, Prof. ND Brunetti, @IngoEitel @t_stiermaier and @ibnsky for this collaboration and for setting up the #GEIST registry on its very beginning - few considerations below 🧵
1/8 Males represent approx. 10% of #Takotsubo patients within the registry. As comapred with females, they have ⬆️comorbid burden (malignancies, COPD etc.), ⬆️physical trigger ⬇️LVEF, worse in-hospital and long-term outcome.
2/8 These results were expected, when looking at smaller previously published studies, but why is that? Is the #takotsubo attack itself worse in men or do they just suffer because of the vulnerable comorbid background?
Our next paper: Abnormal #thyroid function is common in #takotsubo syndrome and depends on two distinct mechanisms: results of a multicentre observational study onlinelibrary.wiley.com/doi/full/10.11…
In this study, we compared patients with #takotsubo syndrome (#TTS) with age- and sex-matched normal subjects from the #NHANES collective and with patients affected by acute coronary syndrome (#ACS) from the AQUA FONTIS study (@StudyAqua). Main outcome measure was serum #FT4. 
With artificial intelligence / machine learning we identified two clusters of #thyroid function in #TTS and three clusters in the whole population. #AI #ML pubmed.ncbi.nlm.nih.gov/33179374/
MINOCA = plaque rupture, plaque erosion, coronary emboli/thrombosis, SCAD, coronary artery spasm, microvascular disease & unclassified MINOCA. #ESCCongress
Diagnostic algorithm for MINOCA: #ESCCongress
Just published on @SciReports: disq.us/t/3qzu3fo,
Role of dyspnea at presentation in #Takotsubo. Thanks to
@FraSantoroMD @beamusumeci1 @lucarlimite @IngoEitel. Here it follows some considerations.
Role of dyspnea at presentation in #Takotsubo. Thanks to
@FraSantoroMD @beamusumeci1 @lucarlimite @IngoEitel. Here it follows some considerations.
1- Dyspnea was associated with both worse in-hospital and long-term outcome. Similarly described in AMI (ref #12 and #13) in which heart failure symptoms at presentation might reflect wider myocardial damage. Why is it so even for a “transient” condition such as #Takotsubo?
2- Dyspnea was independently associated with both higher cardiac impairment and comorbidity burden, intuitively linked to the worse prognosis in the acute phase. And long-term, after recovery of cardiac function? We tried to give two possible explanations (just hypothesis).
Talking with @reepRN and @nsivcd about #Takotsubo cardiomyopathy made me go hunting for an old gif. Clearing out my hard drive so here is an old school gif firesale. Everything must go!
#CardioEd
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