Discover and read the best of Twitter Threads about #IVUS

Most recents (12)

1/ For the fellows and #ACCEarlyCareer!

It’s a coronary thrombus! When to consider thrombectomy? What do you do? Let’s walk through this…#Tweetorial

#Cardiotwitter #Cardiology #STEMI
2/ Middle age patient with hx of CAD and PCI to LAD presents with significant SOB and elevated Hs-Trop. No chest pain. No ECG changes. Echo with inferior hypokinesis.

Here’s the diagnostic with a JR4.

Notice the filling defect in the RCA. This is thrombus. How do we know?
3/ Keys of #thrombus on angiogram

🔑 contrast staining
🔑 Lack of calcium on non con image
🔑 ovoid filling defect (complete lumen)

#Cardiotwitter #STEMI #TIMI
Read 25 tweets
1/25
How would you tackle this heavily calcified LAD with calcified #nodule that was appropriately identified by intravascular imaging? #OCT
doi.org/10.1093/ehjcr/…

In this #EHJCaseReports #tweetorial we take a deep dive into the Rx of #calcified coronary lesions.
#Cardiotwitter
2/25
Calcified lesions are top of the totem pole of complex PCI
Main armamentaria are balloon-based #IVL & ablation-based (rotational/orbital) techniques, while ancillary tools eg guide extension catheters are useful too. eurointervention.pcronline.com/article/intrav…

#EHJCaseReports #tweetorial
3/25
What do you see with this non-compliant balloon in this calcified RCA?
What would you do next? Poll in next tweet! doi.org/10.1093/ehjcr/…
Read 25 tweets
1/ Atypical CLOT- a tweetorial

I recently saw a controversial tweet arguing against tx of chronic clot and restricting tx SOLEY to the CFV/iliac. While I agree (for the most part)- data is limited. At the end of the day we care about the pt, and not every pt is the same! Case➡️ Image
2/ Pathophysiology of chronic #clot formation.

It’s important to recognize the lesions that can form after a clot:

Spurs/Webs
Rokitansky/NIVL/fibrosis

Your pts CEAP >5 always consider for non thrombotic obstructions! Consider #IVUS if things don’t fit! Image
3/ Clot is in a constant state of evolution. #Fibrin—> #Collagen.
 
Unfortunately many patients present in the subacute or late phase of #clot development which hinders many of our treatment approaches.
 
7️⃣days- 20% collagen

1️⃣5️⃣ days- 50% collagen

2️⃣1️⃣days- 80% (!!) collagen Image
Read 19 tweets
3/ Clot is in a constant state of evolution. #Fibrin—> #Collagen.
 
Unfortunately many patients present in the subacute or late phase of #clot development which hinders many of our treatment approaches.
 
7️⃣days- 20% collagen

1️⃣5️⃣ days- 50% collagen

2️⃣1️⃣days- 80% (!!) collagen
Read 39 tweets
1/ Welcome to the first part of a #Tweetorial series on #VenousDisease.

🩸To begin how about we talk about the main player in the game- C.L.O.T. (and why you should care about it)

#Cardiotwitter #Irad #fellows #fellowtwitter #medthread #clot #medtwitter #clottwitter
3/ All clots ARE NOT born equally! There is a difference! Image
Read 34 tweets
AGIKplenum: Unverzichtbar im Katheterlabor!?
(Raum 12 @kardiologie_org)

❤️Erster Vortrag:
Intravaskuläre Bildgebung📸 (David Leistner | @m_leistner | Berlin)

#DGKJahrestagung @HolgerNef @kaschenke @MarcVorpahl @thiele_holger
MLD-MAX Konzept

1: MLD
#DGKJahrestagung 2021
Read 8 tweets
1/24
A picture is worth a thousand words. This month's #EHJCaseReports tweetorial focuses on intravascular imaging in coronary intervention.

For starters, what do you see on this #OCT image of an intracoronary honeycomb🐝?

academic.oup.com/ehjcr/article/…

Poll 👇

#cardiotwitter
2/24
The intracoronary honeycomb on the image above is ⬇️

Find out the answer in #EHJCaseReports:

academic.oup.com/ehjcr/article/…
3/24
Honeycomb-like structure, a rare cause of myocardial ischaemia has multiple communicating channels divided by thin septa. Angiographically fuzzy & hazy appearance often without significant stenosis, they're best detected on high-resolution IVUS/OCT
academic.oup.com/ehjcr/article/…
Read 24 tweets
Check out the #MINOCA section in updated NSTE-ACS guidelines at #ESCcongress
bit.ly/3bcrixp

Here is a tweetorial on #MINOCA
🧲Class I Recommendations🧲

-> Use of Diagnostic algorithm
-> Use of #whyCMR
-> Use of working dx & Rx according to the underlying Dx
MINOCA has MI in it-> DOES the definition allows for the inclusion of non-ischemic causes of troponin elevation?

Given this limitation of the troponin bioassay, the “Fourth 4th Universal of MI" defined Injury from infarction

INJURY IS NECROSIS WITHOUT ISCHEMIA!
#ESCCongress
The hallmark of myocardial injury ⬆️ troponin, However, these entities differ conceptually
▶️ myocardial injury = nonischemic mechanisms of myocyte injury (eg, myocarditis),
▶️MI = ischemic mechanisms (eg,
plaque disruption or supply-demand mismatch).

#ESCCongress
Read 10 tweets
⚡️ Coronary Microvascular Dysfunction is Associated with Significant Plaque Burden & Diffuse Epicardial Atherosclerotic Disease
@JACCJournals

interventions.onlinejacc.org/content/12/15/…
#JACCINT #CMD #CardioTwitter

Let's discuss this paper in 2 Tweets 👇 Image
2/3
First demonstration that majority of pts with #CMD, but with no evidence of significant #CAD by angiography & #FFR, have significant plaque burden by #IVUS ImageImageImageImage
3/3
Take home message:

✅ Large portion of patients with #CMD who have normal looking coronaries or mild #CAD on angiography, have indeed significant atherosclerosis & plaque burden on #IVUS

✅ These findings have important implications in management & prognosis in #CMD ImageImageImageImage
Read 3 tweets
What’s Your “Shear Stress”?

Want to Know More?

Relax & Read ⤵️ #Tweetorial by @CoronaryDoc + @arnavkumar

⚡️Coronary Wall Shear Stress in 12 Tweets ⚡️

1/12
Main Readings:
1) interventions.onlinejacc.org/content/early/…
2) ow.ly/XmD130mc9X8
3) link.springer.com/article/10.100…
#ACCImaging @ACCCardioEd
2/12
Wall shear stress (#WSS) is the frictional force of blood exerted tangential to endothelial surface of vessel wall & expressed in variety of units (1Pa=1N/m2=10 dynes/cm2)

It is derived from
• luminal geometry
• flow rate
• blood viscosity
• near wall velocity values
3/12
In vivo WSS calculations are derived from computational fluid dynamic (#CFD) simulations created from reconstructed 3D vessel geometries (from biplane angio/IVUS/OCT/CT/MR) combined with patient-specific pulsatile inlet/outlet velocity values (Doppler wire or other methods)
Read 12 tweets

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